Additional file 12 of The hibernation-derived compound SUL-138 shifts the mitochondrial proteome towards fatty acid metabolism and prevents cognitive decline and amyloid plaque formation in an Alzheimer’s disease mouse model



Additional file 12: Supplementary dataset 1. differential expression analysis (DEA).
Date made available9-Dec-2023

Keywords on Datasets

  • Alzheimer’s disease
  • APP/PS1 mice
  • Hibernation-derived compound
  • SUL-138
  • Chromanols
  • Long-term potentiation
  • Hippocampus-dependent contextual fear memory
  • Amyloid-beta plaques
  • Mass-spectrometry
  • Mitochondria
  • Fatty acid metabolism

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