Additional file 16 of The hibernation-derived compound SUL-138 shifts the mitochondrial proteome towards fatty acid metabolism and prevents cognitive decline and amyloid plaque formation in an Alzheimer’s disease mouse model

  • Christine de Veij Mestdagh (Creator)
  • Frank T.W. Koopmans (Creator)
  • Jonathan Breiter (Creator)
  • Jaap A Timmerman (Creator)
  • Pieter Vogelaar (Creator)
  • Guido Krenning (Creator)
  • Huibert D. Mansvelder (Creator)
  • August B. Smit (Creator)
  • Rob Henning (Creator)
  • Ronald E. van Kesteren (Creator)

Dataset

Description

Additional file 16: Supplementary dataset 5. GO analysis of hippocampal protein regulation as a result of 3 months of SUL-138 treatment in APP/PS1 mice (APP SUL vs. APP VEH); significantly up regulated group.
Date made available9-Dec-2022
Publisherfigshare

Keywords on Datasets

  • Alzheimer’s disease
  • APP/PS1 mice
  • Hibernation-derived compound
  • SUL-138
  • Chromanols
  • Long-term potentiation
  • Hippocampus-dependent contextual fear memor
  • Amyloid-beta plaques
  • Mass-spectrometry
  • Mitochondria
  • Fatty acid metabolism

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