Additional file 19 of The hibernation-derived compound SUL-138 shifts the mitochondrial proteome towards fatty acid metabolism and prevents cognitive decline and amyloid plaque formation in an Alzheimer’s disease mouse model

  • Christine de Veij Mestdagh (Creator)
  • Frank T.W. Koopmans (Creator)
  • Jonathan Breiter (Creator)
  • Jaap A Timmerman (Creator)
  • Pieter Vogelaar (Creator)
  • Guido Krenning (Creator)
  • Huibert D. Mansvelder (Creator)
  • August B. Smit (Creator)
  • Rob Henning (Creator)
  • Ronald E. van Kesteren (Creator)

Dataset

Description

Additional file 19: Supplementary dataset 8. GO analysis of SUL-138-regulated proteins overlapping with proteins affected in APP/PS1 mice; significantly up regulated group.
Date made available9-Dec-2022
Publisherfigshare

Keywords on Datasets

  • Alzheimer’s disease
  • APP/PS1 mice
  • Hibernation-derived compound
  • SUL-138
  • Chromanols
  • Long-term potentiation
  • Hippocampus-dependent contextual fear memory
  • Amyloid-beta plaques
  • Mass-spectrometry
  • Mitochondria
  • Fatty acid metabolism

Contact

Access Dataset

Cite this