Abstract
Pages F272–F281. van Meurs M, Kurniati NF, Wulfert FM, Asgeirsdottir SA, de Graaf IA, Satchell, SC, Mathieson PW, Jongman RM, Kümpers PK, Zijlstra JG, Heeringa P, Molema G. Shock-induced stress induces loss of microvascular endothelial Tie2 in the kidney which is not associated with reduced glomerular barrier function. Am J Physiol Renal Physiol 297: F272–F281. First published July 23, 2009. doi:10.1152/ajprenal.00137.2009—The above-noted Research Article was published incorrectly.
On p. F273, left column, under materials and methods, Mouse shock models, first full paragraph, the sentence should read as follows.
For the induction of LPS (Escherichia coli, serotype 026:B61; Sigma, St. Louis, MO) at 0.5 μg/g (1,500 endotoxin units/g) endotoxin units/g) body wt 4, 8, and 24 h later, blood was drawn, and organs were harvested as described above.
On p. F279, right column, under discussion the sentence should read as follows. Both studies used C57bL/6 mice, yet the strains of E. coli were different, as was the dose (serotype O55:B5 at 20 mg/kg vs. 026:B6l at 0.5 mg/kg, respectively).
On p. F273, left column, under materials and methods, Mouse shock models, first full paragraph, the sentence should read as follows.
For the induction of LPS (Escherichia coli, serotype 026:B61; Sigma, St. Louis, MO) at 0.5 μg/g (1,500 endotoxin units/g) endotoxin units/g) body wt 4, 8, and 24 h later, blood was drawn, and organs were harvested as described above.
On p. F279, right column, under discussion the sentence should read as follows. Both studies used C57bL/6 mice, yet the strains of E. coli were different, as was the dose (serotype O55:B5 at 20 mg/kg vs. 026:B6l at 0.5 mg/kg, respectively).
Original language | English |
---|---|
Pages (from-to) | F456-F456 |
Number of pages | 1 |
Journal | American journal of physiology-Renal physiology |
Volume | 312 |
Issue number | 3 |
DOIs | |
Publication status | Published - 1-Mar-2017 |