Three strikes to a hemophilic joint bleed

Ton Lisman*

*Corresponding author for this work

Research output: Contribution to journalEditorialAcademicpeer-review

Abstract

In this issue of Blood, Wyseure et al identified that defective activation of thrombin-activatable fibrinolysis inhibitor (TAFI) drives joint bleeding in congenital but not acquired hemophilia A.(1)

Original languageEnglish
Pages (from-to)1548-1550
Number of pages3
JournalBlood
Volume132
Issue number15
DOIs
Publication statusPublished - 11-Oct-2018

Keywords

  • DEPENDENT INHIBITION
  • FIBRINOLYSIS
  • COAGULATION
  • PLASMA
  • THROMBIN
  • ACTIVATION
  • BLOOD
  • TAFI

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