Saved by the Matrix: UPR Independent Survival under ER Stress

Mandy Koopman; Claudio Hetz; Ellen A A Nollen

doi:10.1016/j.cell.2019.11.012

Saved by the Matrix: UPR Independent Survival under ER Stress

Mandy Koopman, Claudio Hetz, Ellen A A Nollen^*

^*Corresponding author voor dit werk

Onderzoeksoutput: Editorial › Academic

8 Citaten (Scopus)

37 Downloads (Pure)

Samenvatting

Cells are protected from endoplasmic reticulum stress through the unfolded protein response (UPR). In this issue of Cell, Schinzel, Higuchi-Sanabria, Shalem et al., identify a mechanism that helps cells cope with ER stress but is independent of canonical UPR activation, instead involving the extracellular matrix hyaluronidase, TMEM2, as a signaling mediator.

Originele taal-2	English
Pagina's (van-tot)	1246-1248
Aantal pagina's	3
Tijdschrift	Cell
Volume	179
Nummer van het tijdschrift	6
DOI's	https://doi.org/10.1016/j.cell.2019.11.012
Status	Published - 27-nov.-2019

Toegang tot document

10.1016/j.cell.2019.11.012Licentie: Elsevier

Saved by the Matrix_ UPR Independent Survival under ER Stress _ Elsevier Enhanced ReaderFinal publisher's version, 1,89 MBLicentie: Taverne

Handle.net

Permanente koppeling

Citeer dit

@article{c78e8ae02f0742919673179e2e0b6934,

title = "Saved by the Matrix: UPR Independent Survival under ER Stress",

abstract = "Cells are protected from endoplasmic reticulum stress through the unfolded protein response (UPR). In this issue of Cell, Schinzel, Higuchi-Sanabria, Shalem et al., identify a mechanism that helps cells cope with ER stress but is independent of canonical UPR activation, instead involving the extracellular matrix hyaluronidase, TMEM2, as a signaling mediator.",

author = "Mandy Koopman and Claudio Hetz and Nollen, {Ellen A A}",

year = "2019",

month = nov,

day = "27",

doi = "10.1016/j.cell.2019.11.012",

language = "English",

volume = "179",

pages = "1246--1248",

journal = "Cell",

issn = "0092-8674",

publisher = "Cell Press",

number = "6",

}

TY - JOUR

T1 - Saved by the Matrix

T2 - UPR Independent Survival under ER Stress

AU - Koopman, Mandy

AU - Hetz, Claudio

AU - Nollen, Ellen A A

PY - 2019/11/27

Y1 - 2019/11/27

N2 - Cells are protected from endoplasmic reticulum stress through the unfolded protein response (UPR). In this issue of Cell, Schinzel, Higuchi-Sanabria, Shalem et al., identify a mechanism that helps cells cope with ER stress but is independent of canonical UPR activation, instead involving the extracellular matrix hyaluronidase, TMEM2, as a signaling mediator.

AB - Cells are protected from endoplasmic reticulum stress through the unfolded protein response (UPR). In this issue of Cell, Schinzel, Higuchi-Sanabria, Shalem et al., identify a mechanism that helps cells cope with ER stress but is independent of canonical UPR activation, instead involving the extracellular matrix hyaluronidase, TMEM2, as a signaling mediator.

U2 - 10.1016/j.cell.2019.11.012

DO - 10.1016/j.cell.2019.11.012

M3 - Editorial

C2 - 31778650

SN - 0092-8674

VL - 179

SP - 1246

EP - 1248

JO - Cell

JF - Cell

IS - 6

ER -

Saved by the Matrix: UPR Independent Survival under ER Stress

Samenvatting

Toegang tot document

Handle.net

Vingerafdruk

Citeer dit